Effects of two black teas, two oolong teas, and two dark teas on liver coefficient, sumption are displayed in Figure 1. Mounting evidence demonstrated that PPARα Inhibitor Compound chronic alcohepatic TG level, and physique weight in mice exposed to long-term excessive alcohol conhol exposure triggered abnormal lipid accumulation and hepatic steatosis, resulting in liver sumption are displayed in Figure 1. Mounting evidence demonstrated that chronic alcohol injury [39]. The model group showed an apparent increase in liver coefficient (p 0.05) and exposure brought on abnormal lipid accumulation and hepatic steatosis, resulting in liver hepatic TG level (p 0.01) compared with all the handle group. Nonetheless, there was no injury [39]. The model group showed an obvious improve in liver coefficient (p 0.05) marked difference in body weight among the model, handle, and all tea extract suppleand hepatic TG level (p 0.01) compared together with the control group. Even so, there was no mentary distinction in physique weight among the 0.05). Incontrol, and all tea extract supplemengroups, as shown in Figure 1C (p model, addition, even though a decreased tenmarked dency in liver coefficient Figure 1C (p 0.05). Additionally, although a decreased tendency tary groups, as shown in was NPY Y1 receptor Antagonist Formulation observed in all tea extract supplementary groups in comparison with all the model group, there all tea extract supplementary groups in(p 0.05). As in liver coefficient was observed in was no marked distinction in Figure 1A comparison observed from Figure 1B, the administration of difference in Tea (OT1), Fenghuang seen from with the model group, there was no marked Tieguanyin Figure 1A (p 0.05). As Danzong Tea (OT2), Fu Brick Tea (DT1), and Selenium-Enriched Dark Tea (DT2) extracts signifiFigure 1B, the administration of Tieguanyin Tea (OT1), Fenghuang Danzong Tea (OT2), cantly inhibited the boost in hepatic TG level induced by chronic alcohol consumption. Fu Brick Tea (DT1), and Selenium-Enriched Dark Tea (DT2) extracts significantly inhibHowever, Dianhong Tea (BT1) levelYingde Black Tea (BT2) did consumption. Nevertheless, ited the boost in hepatic TG and induced by chronic alcohol not considerably inhibit the hepatic Tea accumulation. Dianhong TG (BT1) and Yingde Black Tea (BT2) did not considerably inhibit the hepaticTG accumulation.Figure 1. The effects of teas on (A) liver coefficient, (B) hepatic TG level and (C) physique weight in mice exposed to chronic alcohol consumption. CTRL, the handle group; EtOH, the model group; BT1, Dianhong Tea; BT2, Yingde Black Tea; OT1, Tieguanyin Tea; OT2, Fenghuang Danzong Tea; DT1, Fu Brick Tea; DT2, Selenium-Enriched Dark Tea. p 0.05, p 0.01, the model group compared using the manage group; # p 0.05, ## p 0.01, ### p 0.001, the tea extract supplementary groups compared together with the model group.3.two. Effects of Tea Extracts on Aminotransferase Activities and Serum TG and TC Levels The elevated AST and ALT levels indicated the elevated permeability and harm in liver. As can be noticed from Figure 2A,B, the serum AST and ALT levels in model group were considerably increased to 1.4-fold (181.eight 39.1 vs. 126.7 25.4) (p 0.05) and 1.7-foldgroups compared with all the model group.3.two. Effects of Tea Extracts on Aminotransferase Activities and Serum TG and TC LevelsFoods 2021, ten,The elevated AST and ALT levels indicated the improved permeability and harm 7 of in liver. As can be noticed from Figure 2A,B, the serum AST and ALT levels in model group 25 had been substantially enhanced to 1.4-fold (18.